PubMed
ARA 290, a nonerythropoietic peptide engineered from erythropoietin, improves neuropathic symptoms in patients with sarcoidosis
Dahan et al., 2013
Cibinetide
Non-erythropoietic peptide studied for neuropathic and inflammatory indications.
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Overview
Non-erythropoietic peptide studied for neuropathic and inflammatory indications.
Community
Positive 33% · Neutral 67% · Negative 0%
Median: 2000+ mcg · Most common: 2000+ mcg
Research
PubMed
Dahan et al., 2013
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This page summarizes 6 anonymized self-reports from PeptIQ users who track ARA-290, including commonly reported effects and co-tracked peptides. These are observational patterns, not clinical outcomes.
1 sources are linked on this page, including PubMed articles, clinical trial registries, and FDA labels where applicable. Citations describe published research — not recommendations.
This wiki does not assess safety or recommend use. ARA-290 is listed as Investigational (Phase 2). Consult a licensed clinician for personal medical decisions.
Research, primarily in animal models, suggests ARA-290 may have a wide range of therapeutic potentials due to its ability to promote angiogenesis (formation of new blood vessels), stimulate collagen synthesis, and modulate inflammatory responses.
SourceARA-290 is not approved by the FDA for any human use. There is no legal basis for selling it as a drug, food, or dietary supplement in the United States. The FDA has classified ARA-290 as a Category 2 bulk drug substance, which explicitly prohibits licensed compounding pharmacies from using it in compounded medications.
SourceThe safety and effectiveness of ARA-290 have not been thoroughly evaluated in humans through rigorous clinical trials. This lack of human data means that safe dosages, short-term side effects, and long-term health consequences are largely unknown.
SourceWhile there are over 200 published studies on ARA-290, the vast majority are animal or in vitro (cell) studies. These preclinical studies consistently show positive results across various tissue types. However, there is a significant lack of comprehensive human clinical trial data.
Source