ded for entertainment, informational & educational purposes only & not for the purpose of rendering medical advice, always seek the advice of a health professional

ether or not to store. And there's a gating mechanism, on or off, and GIP hammers the off switch. This is where everybody is missing the whole game. Terzepatide is designed to prevent storage, period. Keeps everything out except what if the problem is what's inside already. And now retatrutide, same thing. GLP-1 and GIP but it adds glucagon agonism. And here's where the instas are confusing everybody because they think glucagon is the same as GLP-1. Not even close. GLP-1 and GIP are satiety molecules. Glucogon is a mobilization molecule. It's about energy extraction. So if you switch from trisepatide to retatrutide and the appetite suppression doesn't hit the same, it's because biology is refusing to listen to the glucagon signal. So here's what's happened. One, the caloric restriction from trisepatide causes biological adaptation and metabolic rate tanks about 20%. Two, caloric restriction sets off this cortisol response. Now pay attention. High cortisol married to low metabolic rate causes hepatic insulin resistance. And your thyroid, well, all of that high cortisol activates 11-beta-HSD1, which cranks up local cortisol in the hypothalamus, which suppresses TRH release. So now you have metabolic stagnation, high cortisol, fatty liver, and suppressed thyroid. And all of this needs to be fixed simultaneously. So when you stop teresapatite, this is the only time I'm going to say this, do a complete water fast, nothing else for 48 hours, water and electrolytes, sodium, potassium, magnesium, and nothing else. Why? Because you need to reset your leptin signaling and give your digestive system a break. 2016 study cell metabolism showed aism, showed a 48-hour fast caused 50% increase in circulating adiponectin levels and normalization of leptin receptor sensitivity. Listen, leptin is the hormone that tells your brain about energy status. If the signaling is broken, and it is if you've been dieting, your brain thinks that you're starving even if you're not. The fast resets this. Now, for two weeks, you eat timed carnivore, 12 calories per pound of target body weight. The first meal at 1 p.m., the second meal at 5. Yes, timing matters. Cortisol peaks at 8 a.m. and tanks by midnight. You're eating when cortisol is naturally lower and doesn't trigger more cortisol spikes. Still not done. For the following two weeks, run the same carnivore diet except add 100 grams of white rice to the first meal and 50 grams of white rice to the second meal. And everybody is going to argue about the white rice except it has the glycemic index necessary to cause the rapid glucose spike. That glucose spike is what triggers hepatic glycogen repletion and triggers T3 production. A 2018 study in Nutrition Reviews showed white rice consumption caused an acute increase in serum T3 concentration of 22% in individuals with suppressed T3 levels. There's a lot more to this. It's all in the research playbook. I don't want anything from you. Just use it. Comment lean for the playbook. I'll also put all of this in my stories at some point this week. I've gotta go. Never miss.